Are you still buying unhealthy BPA leaking SODA bottles?
Now you can stop the problem at it’s roots by making your own soda at home!
The sodastream soda maker let’s you and your family enjoy soda without having BPA’s leak into your body!
This money saving device also helps the environment because it reduces [...]
Are you still buying unhealthy BPA leaking SODA bottles?
Now you can stop the problem at it’s roots by making your own soda at home!
The sodastream soda maker let’s you and your family enjoy soda without having BPA’s leak into your body!
This money saving device also helps the environment because it reduces the electricity used to recycle and reprocess plastic bottles in the first place!
Each BPA-free sodastream bottle is designed to be used thousands of times – so you’ll save your family from ingesting BPA from thousands of soda bottles – many of which are consumed by pregnant women and children – who are the most susceptible.
Why you should eliminate BPA from you, your family and friends (sodastream makes a great gift!)
Biphenol A (BPA) is an endocrine disruptor which can mimic the body’s own hormones and may lead to negative health effects. Early development appears to be the period of greatest sensitivity to its effects, and some studies have linked prenatal exposure to later neurological difficulties.
BPA, like other xenoestrogens, should be considered a player within the nervous system that can regulate or alter its functions through multiple pathways. A 2007 review has concluded that low doses of BPA during development have persistent effects on brain structure, function and behavior in rats and mice.[52] A 2008 review concluded that low-dose BPA maternal exposure causes long-term consequences at the level of neurobehavioral development in mice.[53] A 2008 review has concluded that neonatal exposure to Bisphenol-A (BPA) can affect sexually dimorphic brain morphology and neuronal adult phenotypes in mice.[54] A 2008 review has concluded that BPA altered long-term potentiation in the hippocampus and even nanomolar dosage could induce significant effects on memory processes.[55] A 2009 review raised concerns about BPA effect on anteroventral periventricular nucleus.[56]
A 2005 review concluded that prenatal and neonatal exposure to BPA in mice can potentiate the central dopaminergic systems, resulting in the supersensitivity to the drugs-of-abuse-induced reward effects and hyperlocomotion.[60]
A 2007 review has concluded that bisphenol-A has been shown to bind to thyroid hormone receptor and perhaps have selective effects on its functions.[64]
A 2009 review about environmental chemicals and thyroid function raised concerns about BPA effects on triiodothyronine and concluded that “available evidence suggests that governing agencies need to regulate the use of thyroid-disrupting chemicals, particularly as such uses relate exposures of pregnant women, neonates and small children to the agents”.[65]
A 2009 review summarized BPA adverse effects on thyroid hormone action.[66]
Cancer Research ccording to the WHO’s INFOSAN, carcinogenicity studies conducted under the US National Toxicology Program, have shown increases in leukaemia and testicular interstitial cell tumours in male rats[67]
A 2010 review at Tufts University Medical School concluded that Bisphenol A may increase cancer risk.[68]
Breast cancer
A 2008 review stated that “evidence from animal models is accumulating that perinatal exposure to (…) low doses of (..) BPA, alters breast development and increases breast cancer risk”.[69] Another 2008 review concluded that “animal experiments and epidemiological data strengthen the hypothesis that fetal exposure to xenoestrogens may be an underlying cause of the increased incidence of breast cancer observed over the last 50 years”.[70]
A 2009 in vitro study has concluded that BPA is able to induce neoplastic transformation in human breast epithelial cells.[71] Another 2009 study concluded that maternal oral exposure to low concentrations of BPA during lactation increases mammary carcinogenesis in a rodent model.[72]
A 2010 study with the mammary glands of the offspring of pregnant rats treated orally with 0, 25 or 250 µg BPA/kg body weight has found that key proteins involved in signaling pathways such as cellular proliferation were regulated at the protein level by BPA.[73]
A 2010 study has found that BPA may reduce sensitivity to chemotherapy treatment of specific tumors.[74]
Neuroblastoma
In vitro studies have suggested that BPA can promote the growth of neuroblastoma cells.[75][76] A 2010 in vitro study has concluded that BPA potently promotes invasion and metastasis of neuroblastoma cells through overexpression of MMP-2 and MMP-9 as well as downregulation of TIMP2.[77]
Prostate development and cancer
A 1997 study in mice has found that neonatal BPA exposure of 2 μg/kg increased adult prostate weight.[78] A 2005 study in mice has found that neonatal BPA exposure at 10 μg/kg disrupted the development of the fetal mouse prostate.[79] A 2006 study in rats has shown that neonatal bisphenol A exposure at 10 μg/kg levels increases prostate gland susceptibility to adult-onset precancerous lesions and hormonal carcinogenesis.[80] A 2007 in vitro study has found that BPA within the range of concentrations currently measured in human serum is associated with permanent increases in prostate size.[81] A 2009 study has found that newborn rats exposed to a low-dose of BPA (10 µg/kg) increased prostate cancer susceptibility when adults.[82]
Enough said! Get or Gift A Sodastream!


